Datopotamab Deruxtecan, a Novel TROP2-directed Antibody–drug Conjugate, Demonstrates Potent Antitumor Activity by Efficient Drug Delivery to Tumor Cells

癌症研究 细胞凋亡 拓扑异构酶 细胞培养 药理学 抗体 化学 作用机理 药品 细胞毒性 癌细胞 癌症 DNA损伤 体内 抗体-药物偶联物 细胞 单克隆抗体 前药 医学 抗原 细胞内 肺癌 转染 免疫疗法 程序性细胞死亡
作者
Daisuke Okajima,Satoru Yasuda,Takanori Maejima,Tsuyoshi Karibe,Ken Sakurai,Tetsuo Aida,Tadashi Toki,Junko Yamaguchi,Michiko Kitamura,Reiko Kamei,Tomomichi Fujitani,Tomoyo Honda,Tomoko Shibutani,Sumie Muramatsu,Takashi Nakada,Riki Goto,Shu Takahashi,Miki Yamaguchi,Hirofumi Hamada,Yutaka Noguchi
出处
期刊:Molecular Cancer Therapeutics [American Association for Cancer Research]
卷期号:20 (12): 2329-2340 被引量:314
标识
DOI:10.1158/1535-7163.mct-21-0206
摘要

Abstract Trophoblast cell surface antigen 2 (TROP2) is highly expressed on various epithelial tumors and correlates with poor prognosis. We developed the novel TROP2-directed antibody–drug conjugate (ADC), datopotamab deruxtecan (Dato-DXd, DS-1062a), with a potent DNA topoisomerase I inhibitor (DXd), and evaluated its antitumor activity and safety profiles in preclinical models. The pharmacologic activity and mechanism of action of Dato-DXd were investigated in several human cancer cell lines and xenograft mouse models including patient-derived xenograft (PDX) models. Safety profiles were also assessed in rats and cynomolgus monkeys. Dato-DXd bound specifically to TROP2 and was internalized into tumor cells followed by intracellular trafficking to lysosome and DXd release, which induced DNA damage and apoptosis in TROP2-expressing tumor cells in vitro. Dato-DXd exhibited in vivo antitumor activity with DNA damage induced by the accumulated DXd in TROP2-expressing xenograft tumors, but neither isotype control IgG-ADC nor anti-TROP2 antibody had this effect. Dato-DXd also showed potent antitumor activity with tumor regression in several TROP2-expressing xenograft tumors including NSCLC PDX models. Safety profiles of Dato-DXd in rats and cynomolgus monkeys were acceptable. Dato-DXd demonstrated potent antitumor activity against TROP2-expressing tumors by efficient payload delivery into tumors and acceptable safety profiles in preclinical models. These results suggest Dato-DXd could be a valuable treatment option for patients with TROP2-expressing tumors in the clinical setting.
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