医学
药理学
机制(生物学)
炎症
氧化应激
汤剂
信号转导
作用机理
PI3K/AKT/mTOR通路
肺
细胞损伤
肿瘤坏死因子α
生物信息学
作者
Jiaxin Li,Jiayao Zhao,Zack Shan,Jian Zhang,Mengyan Gong,Qun Liang
标识
DOI:10.2174/0113816128397243250902131510
摘要
This study suggests that the mechanism of BZYQD in treating S-ALI may be achieved by inhibiting the TNF and PI3K signaling pathway and reducing inflammation and oxidative stress levels.
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