Mitogen-Activated Protein Kinase Pathway Activation by the CD6 Lymphocyte Surface Receptor

蛋白激酶A 丝裂原活化蛋白激酶 细胞生物学 淋巴细胞活化 受体 化学 激酶 生物 免疫学 生物化学 免疫系统 T细胞
作者
Anna Ibáñez,Maria‐Rosa Sarrias,Montserrat Farnós,Idoia Gimferrer,Carles Serra‐Pages,Jordi Vives,Francisco Lozano
出处
期刊:Journal of Immunology [American Association of Immunologists]
卷期号:177 (2): 1152-1159 被引量:59
标识
DOI:10.4049/jimmunol.177.2.1152
摘要

CD6 is a cell surface receptor primarily expressed on immature thymocytes and mature T and B1a lymphocytes. Through its binding to activated leukocyte cell adhesion molecule (ALCAM/CD166), CD6 is considered to play an important role in lymphocyte development and activation. Accordingly, CD6 associates with the TCR/CD3 complex and colocalizes with it at the center of the mature immunological synapse on T lymphocytes. Moreover, the CD6-ALCAM interaction has been shown to be critical for proper immunological synapse maturation and T cell proliferative responses. However, the precise biological effects of CD6 ligation and its signaling pathway are still not well understood. The present study shows that CD6 ligation with three different specific mAbs (161.8, SPV-L14.2, and MAE1-C10) induces time- and dose-dependent activation of ERK1/2 on normal and leukemic human T cells. This effect was also observed upon CD6 ligation with a chimerical ALCAM protein (ALCAM-Fc). The C-terminal cytoplasmic region of CD6, as well as Src tyrosine kinases, was critical for CD6-induced ERK1/2 activation. Synergistic effects were observed upon coligation of the TCR/CD3 complex with CD6. The ligation of CD6 induced the transcriptional activation of reporter genes under the control of the c-Fos serum responsive element and AP-1. Accordingly, CD6-mediated activation of p38 and JNK was also observed. These findings indicate that the CD6-ALCAM interaction results in activation of the three MAPK cascades, likely influencing the dynamic balance that determines whether resting or activated lymphocytes survive or undergo apoptosis.
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