APX公司
抗氧化剂
光合作用
超氧化物歧化酶
化学
脂质过氧化
叶绿体
光系统II
生物化学
氧化应激
过氧化物酶
谷胱甘肽
光系统I
谷胱甘肽过氧化物酶
铜毒性
光系统
生物物理学
毒性
生物
酶
有机化学
基因
作者
Fatma Nur,Busra Arikan,Ceyda Ozfidan‐Konakci,Melike Balcı,Evren Yıldıztugay,Halit Çavuşoğlu
标识
DOI:10.1021/acs.jafc.2c02608
摘要
) and lipid peroxidation decreased. CNTs were promoted to the maintenance of the redox state by regulating enzyme/non-enzyme activity/contents involved in the AsA-GSH cycle. Furthermore, CNTs inverted the negative effects of Cu by upregulating the transcriptions of photosystem-related genes. However, the high CNT concentration had adverse effects on the antioxidant capacity. CNT has great potential to confer tolerance by reducing Cu-induced damage and protecting the biochemical reactions of photosynthesis.
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