Emerging Connections Between Inflammatory Bowel Disease Subtypes and Sequential Changes in Total Serum Bilirubin

Lay Summary Building on my long-standing hypothesis that impaired inactivation of digestive proteases by deconjugated bilirubin may be the unifying feature and mechanism of inflammatory bowel disease (IBD), this paper explores potential connections among different subtypes of IBD through sequential changes in total serum bilirubin.