骨愈合
活力测定
细胞生物学
化学
骨形态发生蛋白2
碱性磷酸酶
间充质干细胞
运行x2
MAPK/ERK通路
再生(生物学)
信号转导
生物
细胞
解剖
体外
生物化学
酶
作者
Lei Han,Canfeng Wang,Tuo Wang,Yungeng Hu,Hongshun Wang
摘要
TFRD improves tendon-bone healing in ACLR mouse models and facilitates the osteogenic differentiation of BMSCs through the ERR1/2-Gga1-TGF-β/MAPK pathway, which might deepen our understanding of the underlying mechanism of TFRD in tendon-bone healing.
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